Forensik 1,2

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Asfiksia

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Judicial Hanging

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ETIOLOGI :

1. ALAMIAH :

- PENYAKIT SAL NAAS.

!. MEKANIK :

- T"A#MA

- S#M$ATAN SAL. NAAS. %. KE"ANAN :

- &NS 'EP"ESANT.

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ASE PA'A ASIKSI :

1. 'YSPNOE

!. KON(#LSI

%. APNOE ) AKHI"

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TAN'A-TAN'A ASIKSI PA'A

JENASAH :1. &YANOSIS.

!. LE$AM MAYAT : * LE$IH GELAP

* LE$IH L#AS

* LE$IH &EPAT TE"$ENT#K.

%. $#SAH HAL#S :

- 'EPAN HI'#NG M#L#T

- SAL#"AN NAAS

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TRIAS ASFIKSIA :

busa halusperbendungan

(kongesti)

warna darah lebih gelap

CYANSIS

!"#A$ $AYAT :

lebih gelaplebih luas

lebih %epat terbentuk

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TRIAS ASFIKSIA :


(kongesti)


CYANSIS

!"#A$ $AYAT :



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TRIAS ASFIKSIA :


(kongesti)


CYANSIS

!"#A$ $AYAT :



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+. PELE$A"AN PEM$#L#H 'A"AH

$INTIK! PE"'A"AHAN)TA"'IE#

SPOT)PETE&HIAEL HEMO""HAGE.

,. PE"$EN'#NGAN ) KONGESTI.

. OE'EM P#LMONE".

. 'A"AH LE$IH EN&E" / GELAP.

TAN'A-TAN'A ASIKSI 0&n2.3

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&elebaran &e'buluh

arah

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ede' &ul'onu'

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ASIKSIA MEKANIK

1. PEM$EKAPAN)SMOTHE"ING.

!. GAGGING 4 &HOKING.

%. PEN&EKIKAN.+. PENJE"ATAN ) ST"ANG#LASI.

,. GANT#NG ) HANGING.

. T"A#MATI& ASIKSIA.

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M5kanis65 K56a2ian Pada Asfiksia

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Ana26i l5758

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GANT#NG ) HANGING

9 JEJAS JE"AT

1. MENGA"AH KEATAS KE SIMP#L.

MENGHILANG PA'A $."AM$#T.!. 'IATAS "A;AN GON'OK.

%. SIMP#L HI'#P.

9 LE$AM MAYAT P' #J#NG E

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9 POSISI GANT#NG :

1. KOMPLIT HANGING

!. INKOMPLIT HANGING A. '#'#K) $E"L#T#T

$. $E"$A"ING TE"L#NGK#P.

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9 LETAK SIMP#L :

1. TYPI&AL HANGING: $LK KEPALA

!. ATYPI&AL HANGING : - SAMPING LEHE" KI"I=KANAN

- 'EPAN.

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SE$A$ KEMATIAN

0GANT#NG31. ASIKSIA.

!. ANOKSIA JA"INGAN OTAK.

JE"AT KE&IL4KE"AS= LETAK SIMP#L= POSISI GANT#NG

%. (AGAL "ELE

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Lida7 M5n>ulu8 a2au Tidak?

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Sf2 Liga2u85?

A2au

Ha8d Liga2u85?

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Ha8d Liga2u85

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Sf2 Liga2u85 a2au Ha8d Liga2u85?

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T@ical a2au a2@ical?

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How can someone hang oneself in sitting,kneeling, or lying down positions?

The amount of pressure necessary tocompress :

The jugular veins is 4.4 lb

The carotid arteries, !! lb and

The vertebral arteries, "" lb.

The trachea re#uires $$ lb of pressure.

%nd weight of the head &!'(!) lb* is sufficient toocclude the carotid arteries and cause of death.

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Tehnik topsi

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Tehnik topsi

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e%o'posed bod

PENJE"ATAN )ST"ANG#LASI

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PENJE"ATAN )ST"ANG#LASI

9 JE"AT--- JEJAS JE"AT)SIM P#L

9 JEJAS B L#KA LE&ET TEKAN

1. MEN'ATA" =SEL#"#H LEHE"!. 'I$A;AH "A;AN GON'OK.

%. SIMP#L MATI.

9 JEJAS JE"AT --- TALI PENJE"AT---KE"AS= KE&IL= KASA" ---JELAS

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PENJE"ATAN 0&n2.3

--HAL#S= LE$A". L#NAK ---- T.JELAS

9 ASIKSIA ) (AGAL "ELE

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$anual Strangulation

(&en*eratan)

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Manual S28angula2in

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PEN&EKIKAN

9 L#KA LE&ET KE&IL! $ENT#K $#LAN

SA$IT 'I LEHE" --- K#K#.

9 L#KA MEMA" * K#LIT)OTOT LEHE"

9 PATAH T#LANG LI'AH

9 PATAH T#LANG "A;AN GON'OK

9 PE"$EN'#NGAN *M#KA)KEPALA.9 ASIKSIA ) (AGAL "ELE

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&lasti% #ag Su++o%ation

S

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S'othering

(&e'bekapan)

S'othering

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(&e'bekapan)

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TENGGELAM / DROWNINGTENGGELAM / DROWNING

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TENGGELAM / DROWNINGTENGGELAM / DROWNING

'5f. : Sua2u 8s5s @g di7asilkan l57 258C5na6n@a k8Can dala6 ai8 @ang

65n@5CaCkan k57ilangan k5sada8an 4 dan 65nganca6 >iDa.

'iagnsa ) s58ing suli2 Cila 2ak ada 2anda k7as

T5ngg5la6 daa2 s5lu8u7 2uCu7 ) 6uka 258C5na6

K56a2ian : d2. AkiCa2 25ngg5la6 a2au suda7 6a2i l57 ka85na s5CaC lain.

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PEMERIKSAAN LUAR JENAZAH

Tanda2 terendam dalam air

1.Basa! "erl#m#ran $asir! l#m$#r! dll

2.Tela$a% tan&an ' %a%i %eri$#t

()aser )*man Hand+

,.K#lit $erm#%aan %asar K#lit "e"e%

(-#tis Anserina+

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gambar

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Asphyxia can literally be translatedfrom the +reek as meaning absence ofpulse, but is usually the term given to

deaths due to ano-ia or hypo-ia.The term asphy-ia is thought by someforensic pathologists to be a vague andconfusing term.

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n its broadest sense it refers to astate in which the body becomesdeprived of o-ygen while in e-cess ofcarbon dio-ide &ie. hypo-ia and

hypercapnoea*.

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T75 classic sign f as7@Fia P525c7ial 756887ag5s

&ng5s2in and 5d56a

&@ansis

luidi2@ f 275 Cld

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P525c7ia5 a85 f25n knDn as `tardieu`s spot`,

and disc8iC5d a2 275 fi8s2 2i65 C@ 275 Pa8isian

Professor Ambroise Tardeu in 1/= in 275

Cdi5s f infan2s D7 75 call5d 7ad C55n

58lain

A c66n 5888 is 2 a228iCu25 275 525c7i5 2

275 8u2u85 f cailla8i5s= D7585as 275@ ac2uall@

56ana25 f86 small venules - cailla8@

Cl55ding Duld C5 inisiCl5 2 275 nak5d 5@5.

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P525c7ia5 a85 caus5d C@ an acute rise in

venous pressure that in turn causes over

distension and rupture of thin walled peripheralvenules= s5ciall@ in laF 2issu5s= suc7 as 5@5lid=

and in unsu825d s58us 656C8an5s= suc7

as 275 ic58al lu8a 5s5ciall@ in 275 in258lCa8fissu85s and 5ica8diu6= 27@6us. In 275 C8ain=

525c7ia5 6a@ C5 la8g58 a2c75s f Cl55ding in

275 suCa8ac7nid sac5 D7585 su58ficial5ss5ls 7a5 8u2u85d.

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I2 is 28adi2inall@ clai65d 27a2 7@Fia f 275

5in Dall is an add5d fac28= Cu2 27585 is n

5F58i65n2al 8f f 27is cn>5c2u85= f8

5Fa6l5= 525c7ia5 can a5a8 al6s2ins2an2l@ af258 violent sneezing or coughing

before any hypoxia is possible.

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Ps2u85 als 7as an aff5c2 n 275 a5a8anc5

f 7a56887ag5s. T75@ a85 c66nl@ s55n=

alng Di27 la8g58 5cc7@6s5s= n 275 f8n2 8Cack f c8s5s D7 7a5 di5d f86 a a8i52@

f caus5s in D7ic7 65c7anical as7@Fia is

aCs5n2.

T75@ a85 f25n 85s5n2 in n86al s2 68256

7@s2asis= 5s5ciall@ D7585 275 6d5 f d5a27

Das cng5s2i5 as in 6an@ 2@5s f na2u8al75a82 dis5as5.

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An2758 8Cl56 in 275 au2s@ in25885s2a2in

f 525c7ia5 is 27a2 not all punctate lesions in

the pleura are petechiae. aini and Knig72s7D5d 27a2 6an@ suc7 aa85n2 525c7ia5

D585 5i2758 in28aascula8 5nus ls=

suCl5u8al 6ic8Culla5 8 ig65n2 fci.

In s65 2@5s f d5a27 D7585 F@g5n

d58ia2in is 2 C5 5F5c25d 0suc7 as

d8Dning= las2ic Cag suffca2in and 5n258ingan a26s7585 d5id f F@g5n3= 525c7ia5

a85 seldom demonstrable.

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In su66a8@= 525c7ial 756887ag5s a85 7ig7l@

unreliable indicators of an asphyxial process=

2aking 27is 2 65an a 7@Fic s2a25.

T75@ a85 275 85sul2 f venous engorgement =

usuall@ f86 65c7anical Cs28uc2in f 5nus

852u8n 2 275 75a82 * 8 in 275 278aF f86a22562s 2 insi85 agains2 a Clck5d ai8Da@.

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&ng5s2in and 5d56a

&ng5s2in and 5d56a 27is is 55n 685 nn s5cific27an 525c7ia5 and nc5 again is 275 85sul2 f

Cs28uc25d 5nus 852u8n.

&ng5s2in is f25n asscia25d Di27 2issu5 sD5lling if275 5nus Cs28uc2in cn2inu5s. T75 5d56a is 275

85sul2 f 8aid 28ansuda2in 278ug7 cailla8@ and

5nul5 Dalls= again 6ainl@ a func2in f Cack 85ssu85

in 275 5nus s@s256.

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H@Fia f 275 ascula8 5nd275liu6 is all5g5d 2 allD

inc85as5d 5865aCili2@= Cu2 g5n58ali5d 7@Fia f862758 caus5s d5s n2 8duc5 275 2issu5 sD5lling

s55n in s28angula2in.

Pul6na8@ 5d56a= causing 5Fc5ss fluid 2 5n258 275al5li= is f25n fund in 7@Fic d5a27. H585 275

65c7anis6 is 685 Cscu85= Cu2 i2 is 8CaCl@ a

c6Cina2in f 7@Fia and 8ais5d ul6na8@ 5ss5l

85su85.

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&@ansis

T75 Cluis7 cl8 f s2 68256 liidi2@ d5s n2

7a5 275 sa65 cnn2a2in as c@ansis 8duc5d

du8ing lif5. T75 2586 c@ansis= D7ic7 65ans a

Cluis7 discl8a2in f 275 skin 8 6ucus656C8an5s= s7uld C5 cnfin5d 2 clinical

d5sc8i2ins and n2 us5d f8 c8s5s.

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In 275 liing= 275 c@an2ic cl8 f 275 Cld

85ui85s 275 85s5nc5 f a2 l5as2 , g f 85duc5d7a56glCin 58 c5n2 in 275 cailla8@ Cld.

HD558= in 275 c8s5= F@g5n disscia2in

cn2inu5s and 27585 6a@ C5 85fluF fd5F@g5na25d 5nus Cld in2 275 cailla8i5s.

8 275s5 85asns= 275 Cld f a cada58

C5c65s u8lis7-Clu5= Cu2 27is is n2 275 85sul2f a a277@silgical c7ang5 ccu88ing du8ing

lif5= 5.g. s28angula2in.

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The normal color f a85as f s2 68256 liidi2@ is a

c@an2ic 7u5= Cu2 27is d5sc8i2in s7uld n2 C5 us5d

sinc5 i2 is 6isl5ading.

&@ansis 8duc5d du8ing lif5 6a@ C5 a82l@ 8

D7ll@ 58s7adD5d C@ 7@s2asis= D7ic7 6a@

C5 a d55 u8l5 8 Clu5= and 6a@ C5 6is2ak5nf8 28u5 c@ansis * ind55d= s65 a27lgis2

85fus5 2 us5 c@ansis in 85s5c2 275 d5ad=

clai6ing 27a2 it cannot reflect the ante-mortemsituation.

.

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Documents

Forensik 1,2